Peptide · Research Monograph · Synthetic tuftsin-derived heptapeptide

Selank

Stress Response Studies

Also known as TP-7

Selank is a peptide made of seven amino acids. It is based on tuftsin, a natural immune peptide. It carries the same stabilizing tail used in Semax, and both came out of the same Russian research institute. Studies in rodent models have measured GABA signaling, which is how the brain slows activity down, along with stress-related endpoints.

Formula
C33H57N11O8
Mol. weight
751.88 Da
Length
7 residues
CAS
129954-34-3

For laboratory research use only - not for human or animal use

Selank vial from the Eon Peptides research catalog
Selank · 10mg · catalog photograph

The molecule, in three dimensions

C33H57N11O8751.88
Drag to turn
Residues · hover to locate7 residues
Heavy atoms
53
Bonds
55
Composition
C33 N11 O9
Rings
3
Metal centre
None
Chain length
7 residues

Every heavy atom is placed from the sequence with ideal bond lengths and angles, so the connectivity is exact. The pose is idealised, an extended chain, and is not a measured conformation. Identity for the lot in stock is confirmed by LC-MS on its certificate.

Molecular data

Molecular formulaC33H57N11O8
Molecular weight751.88 Da
SequenceH-Thr-Lys-Pro-Arg-Pro-Gly-Pro-OH
Sequence length7 residues
CAS / identifier129954-34-3
Physical formLyophilized powder
Available sizes10mg

How it works

  1. GABAergic

    GABA-A Receptor Modulation

    Research indicates Selank modulates GABAergic neurotransmission, including GABA-A receptor activity and expression of GABAergic system components. This mechanism is associated with the anxiolytic effects observed in preclinical animal models and early clinical studies, without the sedative or dependency concerns seen with classical benzodiazepines.

    • Modulates GABA-A receptor subunit expression
    • Anxiolytic effects without benzodiazepine-type sedation
    • No reported physical dependence in preclinical models
  2. Neurotrophin

    BDNF & Enkephalin Upregulation

    Raised brain-derived neurotrophic factor (BDNF) expression and altered enkephalin metabolism have been measured. BDNF is central to neuroplasticity and synaptic maintenance. Enkephalin stabilisation is one proposed mechanism behind the mood and cognitive endpoints recorded in preclinical studies.

    • BDNF expression measured in hippocampal regions
    • Stabilizes enkephalin via enkephalinase inhibition
    • Modulates serotonin and dopamine turnover
  3. Immunomodulatory

    Tuftsin-Derived Immune Regulation

    Selank is derived from the immunomodulatory tetrapeptide tuftsin (Thr-Lys-Pro-Arg) with a proline-glycine-proline extension that raises stability. Changes in cytokine production including IL-6, IL-1β and TNF-α expression have been measured, which is why it is investigated for both neurological and immunological activity.

    • Derived from tuftsin (natural immunomodulator)
    • Modulates pro-inflammatory cytokine expression
    • Influences interferon alpha/gamma signaling pathways

What the research shows

  1. Neuropharmacology

    Anxiolytic Mechanisms

    Preclinical studies demonstrate anxiolytic effects in elevated plus maze and open field tests, attributed to GABAergic modulation. Selank shows anxiolytic activity comparable to diazepam without sedative side effects in rodent models.

  2. Cognitive Research

    Memory & Learning

    Learning and memory consolidation measures are recorded in animal models of stress-induced cognitive impairment. Raised BDNF and enkephalin stabilisation are proposed as the underlying mechanisms.

  3. Immunology

    Cytokine & Immune Modulation

    Studies report Selank modulates expression of interleukin-6, TNF-α, and interferon signaling pathways, consistent with its tuftsin-derived immunomodulatory origin. Investigated as a model for peptide-based neuroimmunomodulation research.

  4. Gene Expression

    Transcriptomic Studies

    Gene expression studies identify Selank-induced changes in transcription of genes related to serotonin, dopamine metabolism and synaptic plasticity - a molecular pharmacology framework for studying peptide anxiolytics.

Specification

Chemical NameThr-Lys-Pro-Arg-Pro-Gly-Pro (Selank)
SequenceH-Thr-Lys-Pro-Arg-Pro-Gly-Pro-OH
Molecular Weight751.88 Da
Molecular FormulaC33H57N11O8
FormLyophilized powder
Purity≥99% (HPLC verified)
TestingThird-party HPLC, Mass Spec, Endotoxin
Storage-20°C for long-term stability
SolubilityWater-soluble
COAIncluded with every order

How it's tested

Every lot of Selank released to the catalog carries a third-party certificate of analysis. 3 documented lots are on file for this compound, with reported HPLC purity from 99.61% to 99.75% (mean 99.70%), issued by Accurate Test Labs and Freedom Diagnostics. 2 of 3 certificates record identity as confirmed.

Documented lots
3
Reported purity
99.61% to 99.75%
Mean purity
99.70%
Issuing labs
Accurate Test Labs, Freedom Diagnostics

All certificates in the COA library →

Frequently asked questions

What is Selank and where does it originate?

Selank is a synthetic heptapeptide (7 amino acids) developed at the Institute of Molecular Genetics of the Russian Academy of Sciences. It is derived from the immunomodulatory tetrapeptide tuftsin (Thr-Lys-Pro-Arg), which is a natural fragment of IgG immunoglobulin. A Pro-Gly-Pro extension was added to increase the peptide's stability and half-life. Selank is approved in Russia as an anxiolytic drug for generalized anxiety disorder and neurasthenia.

How does Selank work as an anxiolytic?

Selank's anxiolytic endpoints are attributed to several mechanisms. Changes in GABA-A receptor expression and GABAergic neurotransmission have been measured, similar to but distinct from benzodiazepines. Raised BDNF, enkephalin stabilisation via enkephalinase inhibition, and altered serotonin and dopamine metabolism have also been recorded - a multi-target profile.

Is Selank approved by the FDA?

No. Selank is not approved by the FDA or the European Medicines Agency (EMA). A pharmaceutical form is approved in Russia for anxiety and neurasthenic conditions; the research-grade material sold on eonpeptides.com is not that product. Characterisation outside Russia is very limited, and most research originates from Russian institutions. It is sold here for in vitro research use only.

What makes Selank different from benzodiazepines?

Selank acts on GABAergic activity through a different mechanism from benzodiazepines. In preclinical studies, anxiolytic endpoints were recorded without the sedation, motor impairment or physical dependence seen with classical benzodiazepines. In rodent models it behaves as an anxioselective agent, with anxiety-related behaviour scored separately from sedation.

What is the relationship between Selank and the immune system?

Selank is derived from tuftsin, a natural tetrapeptide with well-characterised immunostimulatory activity. Changes in cytokine production (IL-6, TNF-α, interferon signalling) have been measured, and a neuroimmune bridging role is one proposed interpretation. That dual activity is why it appears in neuroimmunomodulation research on links between anxiety, stress and immune function.

How should research-grade Selank be stored?

Lyophilized Selank should be stored at -20°C away from moisture and light. The Pro-Gly-Pro C-terminal extension raises Selank's resistance to proteolytic degradation compared to tuftsin, giving greater stability in biological research conditions.

For laboratory research use only. Not a drug, supplement, or medical product; not for human or animal use. All findings referenced are from published preclinical/laboratory research.